The highest risk for transsexuality is observed in somatically male individuals carrying a short allele (S) for the ERβ polymorphism together with a G allele for XbaI-ERα and a short allele (S) for AR (SGS genotype) compared to the reference category SAS, short allele (S) for the ERβ together with an A allele for XbaI-ERα and a short allele (S) for AR. However, the differences were not significant when Bonferroni corrections were used
Last sentence... 🤦🏻♀️
When we bootstrapped it the significance disappeared 
I dunno about this one (first paper in the list.) it’s saying there’s a correlation between one variant in one type of hormone receptor and abnormal gender identity. However it’s also saying that in some cases that significance disappears when you look at it using other statistical methods. Hmmm.
It’s possible (actually probably IMO) that there is a biological basis for all the dysphoric behaviours. however I would be very sceptical of ‘a gene for being trans.’
I’ve said it before but to me the most likely mechanism is going to be a general set of issues that lead to an increased likelihood of dysphoric behaviour. These may overlap with other conditions (for example the brain scans have shown some hints that areas implicated in fragile sense of self in schizophrenia are also involved.) so there is probably a biological predisposition to dysphoria.
The form that disphoria takes however i think is HUGELY affected by the societal conditions the person finds themselves in.
All of this applies only to genuine gender dysphoria - Not AGP.
It’s be interesting if a set of strong markers for gender dysphoria were found. I can imagine it being strongly contested by the rest of the umbrella...