Ladyfanny, I appreciate you do not want to argue about CF specifically in a general discussion (although you did bring it up) but I would hope you appreciate also that I have picked up on your comments because it is a matter close to my heart.
my statement was in response to one of your earlier posts saying ?Woman A has a copy of a CF mutation but Child B (who is not her child) has a mother who does not carry a copy, while her husband does. This means that because Child B only inherits one copy of the gene, they will live a CF free life. Until Woman A comes along with her breast milk and transfers this copy of the CF mutation. Child B now has two copies of the gene, and therefore, will develop CF?.
So yes you did not say ?catch? (in the sense of contagious diseases) but ?transfer' but your statement made me so cross that catch and transfer meant the same thing to me
There is anecdotal evidence that carriers of CF can display mild CF like symptoms. Ask any number of parents of children with CF and you will find a high incidence of bowel problems, chest infections, asthma, sinusitis and heavy sweating etc. Bearing in mind there are so few people who actually know they are carriers it is hard to say whether there is any real connection between symptoms and carrier status. It has been suggested that the CFTR function works well enough in a carrier for them to have ?normal? body functions but in some individuals it might not work quite well enough hence the incidence of CF type symptoms.
This, as far as I can see is again down to the genes a person inherits though and not as a result of the interference of someone else?s breastmilk
I don?t know about HIV in any detail but am not sure of the relevance of comparing it to CF as they have very different causes